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Rosetta Stone Arabic free [复制链接]

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发表于 2011-9-1 19:50:47 |只看该作者 |倒序浏览
According to the existing model cholesterol is supposed to be the causing factor of arteriosclerosis. Statins reduce cholesterol in the blood thus protecting from cardiovascular disease. Well,language learning Spanish, none of the above is true anymore. Statins have failed to protect patients from heart disease and cholesterol is now found to be minimally correlated with arteriosclerosis, literally insignificant from a statistical point of view. Cholesterol is not the cause of cardiovascular disease anymore.

Atherosclerosis is tightly associated with homocysteine levels. In fact, in human studies, elevation of blood homocysteine is found to be a greater risk factor (22-40 fold) than elevated cholesterol (1.2 ? 3.1 fold),greek language learning software, high blood pressure (8-18 fold) or cigarette smoking (3.5 fold). It is pretty obvious that Cholesterol scores really low here. But it is the nig pharmaceutical companies' interests that are best served in this way. Selling statins like Lipitor, Crestor etc, result in billions of profit each year for the pharma companies.

In 1968 a grand medical researcher from Harvard Medical School, clarified the atherosclerosis mechanism. His opinions and publications were ignored by the mainstream medical society. Few scientists supported him and it took almost two decades for more official publications,Rosetta Stone Arabic free, except for his, to start supporting his findings. This is a well documented and more than sufficiently supported theory now and clinical and experimental data clearly point towards that direction. Unfortunately it will probably take a couple more decades for the medical society to catch up and incorporate these valuable findings to practice.

It?s all about homocysteine then. There are certain conditions (very common indeed) that this molecule cannot be metabolised properly, it accumulates in the liver and reacts with LDL. The modified LDL is highly toxic and when it is taken in by arterial cells it causes endothelial damage. This is the beginning of arterioslcerosis. Cholesterol is accumulated there as a byproduct of the numerous reactions taking place in the area of endothelial dysfunction. Soon platelets start to accumulate, get stuck to the atheromatic plaques being formed and thrombosis becomes an extra clinical symptom of atherosclerosis. In this way you have the full spectrum of clinical symptoms. If this sequence of events involves the arteries supplying the heart with blood, coronary artery disease is the result and the risk of myocardial infarction is highly elevated. Other parts of the circulatory system involved will equally threaten other vital organs, such as the brain, threatening with stroke,learn spanish, kidneys, threatening with kidney failure and the list goes on?

The bottom-line is that there is a cheap, safe and massively efficient atherosclerosis treatment. Vitamins. Yes, it only takes vitamins B12, B6 and folic acid (folate) to help the liver detoxify the excess of homocysteine. This is why this whole thing started. Severe deficiencies in these vitamins in the first place caused the liver to be unable to metabolize the excessive amount of homocysteine. These vitamins are key factors in the liver detoxification enzymes. Given that a healthy liver is available, supplementation with the missing nutrients provided with instant alleviation of the atherosclerosis condition and gradually reverses the clinical picture. It is unethical for medicine to discount such pioneering research and instead of supporting safe and efficient therapies, prescribes toxic and dangerous statin drugs. Statins have been insufficiently tested on humans before they were released in the market. Taking that into account,Rosetta Stone English, it is no surprise they have horrific sideeffects and they fail 100% in protecting patients from cardiovascular disease. People should know about this. It is a crime committed by pharmaceutical companies and people are paying the price by compromising their lives.
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